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Open Access

Peer-reviewed

Research Article

Human A53T α-Synuclein Causes Reversible Deficits in Mitochondrial Function and Dynamics in Primary Mouse Cortical Neurons

  • Li Li,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

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  • Sashi Nadanaciva,

    Affiliation Compound Safety Prediction Group, Pfizer, Inc., Groton, Connecticut, United States of America

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  • Zdenek Berger,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

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  • Wei Shen,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

    ⨯
  • Katrina Paumier,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

    ⨯
  • Joel Schwartz,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

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  • Kewa Mou,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

    ⨯
  • Paula Loos,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

    ⨯
  • Anthony J. Milici,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

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  • John Dunlop,

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

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  • Warren D. Hirst

    warren.hirst@pfizer.com

    Affiliation Neuroscience Research Unit, Pfizer, Inc., Cambridge, Massachusetts, United States of America

    ⨯

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