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Rapamycin Reverses Status Epilepticus-Induced Memory Deficits and Dendritic Damage

  • Amy L. Brewster,

    Affiliation Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America

  • Joaquin N. Lugo,

    Affiliation Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America

  • Vinit V. Patil,

    Affiliation Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America

  • Wai L. Lee,

    Affiliation Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America

  • Yan Qian,

    Affiliation Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America

  • Fabiola Vanegas,

    Affiliation Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America

  • Anne E. Anderson

    annea@bcm.edu

    Affiliations Cain Foundation Laboratories, Jan and Dan Duncan Neurological Research Institute at Texas Children’s Hospital and Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America, Department of Neurology, Baylor College of Medicine, Houston, Texas, United States of America, Department of Neuroscience, Baylor College of Medicine, Houston, Texas, United States of America

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