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Open Access

Peer-reviewed

Research Article

STAT3 Is Activated by JAK2 Independent of Key Oncogenic Driver Mutations in Non-Small Cell Lung Carcinoma

  • Brendan D. Looyenga ,

    brendan.looyenga@vai.org (BDL); jeff.mackeigan@vai.org (JPM)

    Affiliation Systems Biology, Van Andel Research Institute, Grand Rapids, Michigan, United States of America

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  • Danielle Hutchings,

    Affiliation Systems Biology, Van Andel Research Institute, Grand Rapids, Michigan, United States of America

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  • Irene Cherni,

    Affiliation Lung Cancer Unit, Cancer & Cell Biology Division, The Translational Genomics Research Institute (TGen), Phoenix, Arizona, United States of America

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  • Chris Kingsley,

    Affiliation Diabetes, Cardiovascular & Metabolic Diseases Division, TGen, Phoenix, Arizona, United States of America

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  • Glen J. Weiss ,

    Contributed equally to this work with: Glen J. Weiss, Jeffrey P. MacKeigan

    Affiliations Lung Cancer Unit, Cancer & Cell Biology Division, The Translational Genomics Research Institute (TGen), Phoenix, Arizona, United States of America, Virginia G. Piper Cancer Center Clinical Trials, Scottsdale Healthcare, Scottsdale, Arizona, United States of America

    ⨯
  • Jeffrey P. MacKeigan

    Contributed equally to this work with: Glen J. Weiss, Jeffrey P. MacKeigan

    brendan.looyenga@vai.org (BDL); jeff.mackeigan@vai.org (JPM)

    Affiliation Systems Biology, Van Andel Research Institute, Grand Rapids, Michigan, United States of America

    ⨯

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