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Excess Circulating Alternatively Activated Myeloid (M2) Cells Accelerate ALS Progression While Inhibiting Experimental Autoimmune Encephalomyelitis

  • Ilan Vaknin ,

    Contributed equally to this work with: Ilan Vaknin, Gilad Kunis

    Affiliation Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel

  • Gilad Kunis ,

    Contributed equally to this work with: Ilan Vaknin, Gilad Kunis

    Affiliation Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel

  • Omer Miller,

    Affiliation Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel

  • Oleg Butovsky,

    Affiliation Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel

  • Shay Bukshpan,

    Affiliation Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel

  • David R. Beers,

    Affiliation Department of Neurology, Methodist Neurological Institute, The Methodist Hospital Research Institute, The Methodist Hospital, Houston, Texas, United States of America

  • Jenny S. Henkel,

    Affiliation Department of Neurology, Methodist Neurological Institute, The Methodist Hospital Research Institute, The Methodist Hospital, Houston, Texas, United States of America

  • Eti Yoles,

    Affiliation NeuroQuest Ltd. Misgav Venture Accelerator, Misgav Business Park, Misgav, Israel

  • Stanley H. Appel ,

    michal.schwartz@weizmann.ac.il (MS); sappel@tmhs.org (SHA)

    These authors also contributed equally to this work.

    Affiliation Department of Neurology, Methodist Neurological Institute, The Methodist Hospital Research Institute, The Methodist Hospital, Houston, Texas, United States of America

  • Michal Schwartz

    michal.schwartz@weizmann.ac.il (MS); sappel@tmhs.org (SHA)

    These authors also contributed equally to this work.

    Affiliation Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel

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