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Research Article

Cardiomyocyte Specific Deletion of Crif1 Causes Mitochondrial Cardiomyopathy in Mice

  • Juhee Shin,

    Affiliations: Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea, Department of Life Sciences, Pohang University of Science and Technology, Pohang, Kyungbuk, Republic of Korea

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  • Seok Hong Lee,

    Affiliation: Division of Endocrinology and Metabolism, Department of Internal Medicine, College of Medicine, Chung-Ang University, Dongjak-gu, Seoul, Republic of Korea

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  • Min-Chul Kwon,

    Affiliation: Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea

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  • Dong Kwon Yang,

    Affiliation: Global Research Laboratory and Department of Life Science, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea

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  • Ha-Rim Seo,

    Affiliation: Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea

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  • Jaetaek Kim,

    Affiliation: Division of Endocrinology and Metabolism, Department of Internal Medicine, College of Medicine, Chung-Ang University, Dongjak-gu, Seoul, Republic of Korea

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  • Yoon-Young Kim,

    Affiliations: Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea, Department of Life Sciences, Pohang University of Science and Technology, Pohang, Kyungbuk, Republic of Korea

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  • Sun-Kyoung Im,

    Affiliations: Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea, Department of Life Sciences, Pohang University of Science and Technology, Pohang, Kyungbuk, Republic of Korea

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  • Evan Dale Abel,

    Affiliation: Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, Utah, United States of America

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  • Kyong-Tai Kim,

    Affiliation: Department of Life Sciences, Pohang University of Science and Technology, Pohang, Kyungbuk, Republic of Korea

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  • Woo Jin Park,

    Affiliation: Global Research Laboratory and Department of Life Science, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea

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  • Young-Yun Kong mail

    ykong@snu.ac.kr

    Affiliation: Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea

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  • Published: January 04, 2013
  • DOI: 10.1371/journal.pone.0053577

About the Authors

Juhee Shin, Min-Chul Kwon, Ha-Rim Seo, Yoon-Young Kim, Sun-Kyoung Im, Young-Yun Kong
Department of Biological Sciences, Seoul National University, Gwanak-gu, Seoul, Republic of Korea
Juhee Shin, Yoon-Young Kim, Sun-Kyoung Im, Kyong-Tai Kim
Department of Life Sciences, Pohang University of Science and Technology, Pohang, Kyungbuk, Republic of Korea
Seok Hong Lee, Jaetaek Kim
Division of Endocrinology and Metabolism, Department of Internal Medicine, College of Medicine, Chung-Ang University, Dongjak-gu, Seoul, Republic of Korea
Dong Kwon Yang, Woo Jin Park
Global Research Laboratory and Department of Life Science, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea
Evan Dale Abel
Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, Utah, United States of America

Corresponding Author

Email: ykong@snu.ac.kr

Competing Interests

The authors have declared that no competing interests exist.

Author Contributions

Conceived and designed the experiments: JS Y.Y. Kong. Performed the experiments: JS SHL DKY. Analyzed the data: JS SHL MCK HRS JK DKY Y. Y. Kim SKI EDA KTK Y.Y. Kong. Contributed reagents/materials/analysis tools: WJP JK. Wrote the paper: JS Y.Y. Kong.