Defects in Mitochondrial Dynamics and Metabolomic Signatures of Evolving Energetic Stress in Mouse Models of Familial Alzheimer's Disease

• Eugenia Trushina ,

  * E-mail: Trushina.eugenia@mayo.edu

  Affiliation Department of Molecular Pharmacology and Experimental Therapeutics and Neurology, Mayo Clinic, Rochester, Minnesota, United States of America

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• Emirhan Nemutlu,

  Affiliation Cardiovascular Diseases, Mayo Clinic, Rochester, Minnesota, United States of America

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• Song Zhang,

  Affiliation Cardiovascular Diseases, Mayo Clinic, Rochester, Minnesota, United States of America

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• Trace Christensen,

  Affiliation Electron Microscopy Core Facility, Mayo Clinic, Rochester, Minnesota, United States of America

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• Jon Camp,

  Affiliation Biomedical Imaging Resource, Mayo Clinic, Rochester, Minnesota, United States of America

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• Janny Mesa,

  Affiliation Department of Molecular Pharmacology and Experimental Therapeutics and Neurology, Mayo Clinic, Rochester, Minnesota, United States of America

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• Ammar Siddiqui,

  Affiliation Department of Molecular Pharmacology and Experimental Therapeutics and Neurology, Mayo Clinic, Rochester, Minnesota, United States of America

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• Yasushi Tamura,

  Affiliation Department of Cell Biology, Johns Hopkins University School of Medicine, Baltimore, Maryland, United States of America

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• Hiromi Sesaki,

  Affiliation Department of Cell Biology, Johns Hopkins University School of Medicine, Baltimore, Maryland, United States of America

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• Thomas M. Wengenack,

  Affiliation Departments of Neurology and Biochemistry and Molecular Biology, Mayo Clinic, Rochester, Minnesota, United States of America

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• Petras P. Dzeja,

  Affiliation Cardiovascular Diseases, Mayo Clinic, Rochester, Minnesota, United States of America

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• Joseph F. Poduslo

  Affiliation Departments of Neurology and Biochemistry and Molecular Biology, Mayo Clinic, Rochester, Minnesota, United States of America

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