Lack of nAChR Activity Depresses Cochlear Maturation and Up-Regulates GABA System Components: Temporal Profiling of Gene Expression in α9 Null Mice

Open Access

Peer-reviewed

Research Article

Sevin Turcan,

Affiliations Department of Biomedical Engineering, Tufts University, Medford, Massachusetts, United States of America, Department of Neuroscience, Tufts University School of Medicine, Boston, Massachusetts, United States of America
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Donna K. Slonim,

Affiliation Department of Computer Science, Tufts University, Medford, Massachusetts, United States of America
⨯
Douglas E. Vetter

* E-mail: douglas.vetter@tufts.edu

Affiliation Department of Neuroscience, Tufts University School of Medicine, Boston, Massachusetts, United States of America
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Reader Comments

GABABβ1 subunit at the transcript level. Loss of GABABβ1
http://plosone.org/article/info:doi/10.1371/journal.pone.0009058#article1.body1.sec3.sec3.p3

The corrected text should read: "...GABA(B1) subunit at the transcript level. Loss of GABA(B1)..."

No competing interests declared.

Gene expression
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Cochlea
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Synapses
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Cholinergics
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Microarrays
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Nicotinic acetylcholine receptors
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Immunoprecipitation
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CREB signaling
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